DEPARTMENT OF PSYCHIATRY
Center for Weight and Eating Disorders
Lucy F. Faulconbridge, Ph.D.
Lucy F. Faulconbridge, Ph.D. is a Research Associate at the University of Pennsylvania School of Medicine in the Center for Weight and Eating Disorders. She received her B.A. from the University of St Andrews in Scotland in 2000 and her M.A. and Ph.D. in Clinical Psychology from the University of Pennsylvania in 2002 and 2007, respectively.
Dr. Faulconbridge’s research interests currently focus on the co-morbidity of obesity with mental illness, particularly depression. She is interested in changes in mood following weight reduction, as well as the relationship between weight change and risk factors for cardiovascular disease. Dr. Faulconbridge also has expertise in the endocrine and psychological outcomes following bariatric surgery and non-surgical weight loss, particularly the amelioration of diabetes following surgery and possible hormonal correlates for this. Currently, Dr. Faulconbridge is overseeing an NIH-funded study examining the impact of binge eating disorder on weight loss in bariatric surgery patients and patients enrolled in a behavioral weight loss program. In her clinical work, Dr. Faulconbridge uses cognitive-behavioral therapy to treat patients with eating disorders and obese patients seeking weight loss.
Dr. Faulconbridge can be contacted at: University of Pennsylvania School of Medicine, 3535 Market St., Suite 3021, Philadelphia, PA 19104-3309, 215-898-7314. Her e-mail address is lucyhf@mail.med.upenn.edu.
Recent publications include:
Faulconbridge, LF, Wadden, TA, Berkowitz, RI, Sarwer, DB, Womble, LG, Hesson, LA, Stunkard, AJ, Fabricatore, AN (in press). Changes in Symptoms of Depression with Weight Loss: Results of a Randomized Trial. Obesity.
Sarwer, DB, Fabricatore, AN, Jones-Corneille, L, Allison KC, Faulconbridge, LF, Wadden, TA. (in press) Psychosocial Outcomes Following Bariatric Surgery Primary Psychiatry.
Faulconbridge, LF, Grill, HJ, Kaplan, JM, Daniels, D. (2008) Caudal brainstem delivery of ghrelin induces fos expression in the nucleus of the solitary tract, but not in the arcuate or paraventricular nuclei of the hypothalamus Brain Research 1218: 151-57.
Wadden TA, Butryn ML, Sarwer DB, Fabricatore AN, Crerand CE, Lipschutz PE, Faulconbridge L, Raper S, Williams NN. Comparison of psychosocial status in women with class III versus class I-II obesity. Obesity 2006; 14 (Suppl 2): 90S-98S.
Faulconbridge, L., Grill, H & Kaplan, J. (2005). Reversal of ghrelin hyperphagia with NPY Y1 and NPY Y5 receptor antagonists: differential effects in the forebrain and brainstem. Diabetes 54(7): 1985-93.
Faulconbridge, L., Cummings, E., Kaplan, J., & Grill, H. (2003) Hyperphagic effects of brainstem ghrelin administration Diabetes, 52(9): 2260-5.