Bassil Kublaoui, MD PhD

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Assistant Professor of Clinical Pediatrics
Department: Pediatrics

Contact information
Division of Endocrinology and Diabetes
The Children's Hospital of Philadelphia
34th and Civic Center Blvd, 11NW30
Philadelphia, PA 19104
Philadelphia, PA 19104
Office: 215-590-3174
Fax: 215-590-3053
Education:
BA (Psychology)
Rutgers University, 1990.
BA (Biochemistry)
Rutgers University, 1990.
PhD (Biochemistry)
Boston University School of Medicine, 1997.
MD (Medicine)
Boston University School of Medicine, 1997.
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Description of Research Expertise

My former lab research focused on the hypothalamic regulation of feeding and energy expenditure. I am currently collaborating with Dr. Struan Grant on his project looking at genetics of obesity.

Selected Publications

Lai M, Roizen J, Kublaoui B: Exenetide (Byetta) Prevents Obesity in Mouse Model of Hypothalamic Obesity. Pediatric Academic Society and Pediatric Endocrine Society Annual Meeting, Vancouver, BC May 2014.

Iaumi K, Housam R, Kapadia C, Stallings VA, Medne L, Shaikh TH, Kublaoui BM, Zackai EH, Grimberg A: Endocrine phenotype of 6q16.1-q21 deletion involving SIM1 and Prader-Willi syndrome-like features. Am J Med Genet A 161A(12): 3137-43, Dec 2013.

Xi D, Roizen J, Lai M, Gandhi N, Kublaoui B: Paraventricular nucleus Sim1 neuron ablation mediated obesity is resistant to high fat diet. PLoS One 8(11): e81087, Nov 19 2013.

Kublaoui BM, Levine MA: Receptor Transduction of Hormone Action. Sperling Pediatric Endocrinology. 4th edition. Mark Sperling (eds.). Elsevier, 2013.

Xi, D, Gandhi, N. Lai, M., Kublaoui BM. : Ablation of Sim1 Neurons Causes Hyperphagia, Reduced Energy Expenditure and Obesity. Pediatric Academic Societies Annual Meeting Boston 2012.

Xi, D, Gandhi, N. Lai, M., Kublaoui BM. : Ablation of Sim1 Neurons Causes Hyperphagia, Reduced Energy Expenditure and Obesity. Pediatric Endocrine Society Annual Meeting Philadelphia 2012.

Xi Dong, Gandhi Nilay, Lai Meizan, Kublaoui Bassil M: Ablation of Sim1 neurons causes obesity through hyperphagia and reduced energy expenditure. PloS one 7(4): e36453, 2012.

Xi, D, Gandhi, N. Lai, M., Kublaoui BM. : Hyperphagic obesity, reduced energy expenditure, disrupted MC4R signaling, and abnormal response to high fat diet after ablation of PVH Sim1 neurons. Society for Neuroscience annual meeting 2012.

Tolson Kristen P, Gemelli Terry, Gautron Laurent, Elmquist Joel K, Zinn Andrew R, Kublaoui Bassil M: Postnatal Sim1 deficiency causes hyperphagic obesity and reduced Mc4r and oxytocin expression. The Journal of neuroscience : the official journal of the Society for Neuroscience 30(10): 3803-12, Mar 2010.

Shah Shuchi, Kublaoui Bassil M, Oden Jon D, White Perrin C: Screening for type 2 diabetes in obese youth. Pediatrics 124(2): 573-9, Aug 2009.

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Last updated: 09/17/2014
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