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J. Eric Russell, M.D.
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Associate Professor of Medicine (Hematology-Oncology)
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Department: Medicine
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Contact information
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Biomedical Research Building, Room 713
36 421 Curie Boulevard
Philadelphia, PA 19104
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36 421 Curie Boulevard
Philadelphia, PA 19104
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Office: 215-898-4406
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32 Fax: 215-573-7039
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Email:
JERUSS@MAIL.MED.UPENN.EDU
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JERUSS@MAIL.MED.UPENN.EDU
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Publications
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Education:
21 9 A.B. 27 (Chemistry: magna cum laude) c
39 Harvard University Cambridge, MA, 1980.
21 9 M.D. 15 (Medicine) c
2b Stanford University School of Medicine 24 Palo Alto, CA , 1984.
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Permanent link21 9 A.B. 27 (Chemistry: magna cum laude) c
39 Harvard University Cambridge, MA, 1980.
21 9 M.D. 15 (Medicine) c
2b Stanford University School of Medicine 24 Palo Alto, CA , 1984.
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3d Control and function of human embryonic globin genes.
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58 Key words: Globin, embryonic, sickle cell anemia, thalassemia, mRNA, prothrombin.
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Description of Research Expertise
24 RESEARCH INTERESTS8
3d Control and function of human embryonic globin genes.
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58 Key words: Globin, embryonic, sickle cell anemia, thalassemia, mRNA, prothrombin.
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138 Jiang, Y., Xu, X.-S., and Russell, J.E.: The high stability of human [beta]-globin mRNA is determined by a specific cis-element within its 3'UTR. 28th Annual Meeting of the National Sickle Cell Disease Program, Cincinnati, April 9-13 2005.
13c He, Z., and Russell, J.E.: Evidence that differential stabilization of [beta]-like globin mRNAs is mediated by specific determinants in their 3'UTRs. 28th Annual Meeting of the National Sickle Cell Disease Program, Cincinnati, April 9-13 2005.
13c Russell, J.E.: Adaptive post-transcriptional processes mitigate globin chain imbalance in [beta]-thalassemic erythrocytes. 27th Annual Meeting of the National Sickle Cell Disease Program, Los Angeles, April 18-21 2004 Notes: Oral presentation.
147 He, Z., and Russell, J.E.: Co-expression of human [zeta] globin corrects the sickle phenotype in mouse models of sickle cell disease. 27th Annual Meeting of the National Sickle Cell Disease Program, Los Angeles, April 18-21 2004 Notes: Oral presentation.
124 Frehm, E.J., Russell, J.E., and Gow, A.J.: Genetic and pharmacologic modulation of nitric oxide-hemoglobin reactivity. Eastern Society for Pediatric Research, Old Greenwich CT, March 26-28 2004 Notes: Oral presentation.
66 Russell, J.E.: Hemoglobinopathies and thalassemias. 6e Medical Knowledge Self-Assessment Program 13. P.E. Epstein (eds.). Page: 30-32, 2003.
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Selected Publications
140 Jiang, Y., Xu, X.-S., and Russell, J.E.: The high stability of [beta]-globin mRNA requires site-specific binding of factors that remodel the structure of its 3'UTR. The 8th Cooley's Anemia Symposium, New York Academy of Sciences, March 17-19 2005.138 Jiang, Y., Xu, X.-S., and Russell, J.E.: The high stability of human [beta]-globin mRNA is determined by a specific cis-element within its 3'UTR. 28th Annual Meeting of the National Sickle Cell Disease Program, Cincinnati, April 9-13 2005.
13c He, Z., and Russell, J.E.: Evidence that differential stabilization of [beta]-like globin mRNAs is mediated by specific determinants in their 3'UTRs. 28th Annual Meeting of the National Sickle Cell Disease Program, Cincinnati, April 9-13 2005.
13c Russell, J.E.: Adaptive post-transcriptional processes mitigate globin chain imbalance in [beta]-thalassemic erythrocytes. 27th Annual Meeting of the National Sickle Cell Disease Program, Los Angeles, April 18-21 2004 Notes: Oral presentation.
147 He, Z., and Russell, J.E.: Co-expression of human [zeta] globin corrects the sickle phenotype in mouse models of sickle cell disease. 27th Annual Meeting of the National Sickle Cell Disease Program, Los Angeles, April 18-21 2004 Notes: Oral presentation.
124 Frehm, E.J., Russell, J.E., and Gow, A.J.: Genetic and pharmacologic modulation of nitric oxide-hemoglobin reactivity. Eastern Society for Pediatric Research, Old Greenwich CT, March 26-28 2004 Notes: Oral presentation.
66 Russell, J.E.: Hemoglobinopathies and thalassemias. 6e Medical Knowledge Self-Assessment Program 13. P.E. Epstein (eds.). Page: 30-32, 2003.
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