Michal A. Elovitz, M.D.
Associate Professor of Obstetrics and Gynecology
Department: Obstetrics and Gynecology
Graduate Group Affiliations
Contact information
University of Pennsylvania School of Medicine
Center for Research in Reproduction and Women's Health
421 Curie Blvd., 1354 BRB II/III
Philadelphia, PA 19104 USA
215-573-0859 ph, 215-573-5408 fx
melovitz@obgyn.upenn.edu
,
Center for Research in Reproduction and Women's Health
421 Curie Blvd., 1354 BRB II/III
Philadelphia, PA 19104 USA
215-573-0859 ph, 215-573-5408 fx
melovitz@obgyn.upenn.edu
,
Office: 215-573-0859
Fax: 215-573-5408
Fax: 215-573-5408
Email:
melovitz@obgyn.upenn.edu
melovitz@obgyn.upenn.edu
Publications
Links
Search PubMed for articles
Center for Research on Reproduction and Women's Health Faculty
Penn Health Profile
Search PubMed for articles
Center for Research on Reproduction and Women's Health Faculty
Penn Health Profile
Education:
BA (History/Pre-med)
University of Texas at Austin, Texas (with honors), 1990.
MD (Medicine)
Albert Einstein College of Medicine Bronx, New York, 1994.
Permanent linkBA (History/Pre-med)
University of Texas at Austin, Texas (with honors), 1990.
MD (Medicine)
Albert Einstein College of Medicine Bronx, New York, 1994.
Description of CVI Expertise
CVI Program Unit(s):Lipid / Atherosclerosis / CAD / ACS / Prevention
CVI Research Description:
We hypothesize that preeclampsia, a disease unique to pregnancy and characterized by hypertension, proteinuria and edema, is an early manifestation of cardiovascular disease. Our research focuses on the role of environmental and genetic factors, known to be risk factors or mechanistically involved in cardiovascular disease, in preeclampsia. We are specifically interested in elucidating the contribution of the components of metabolic syndrome to the pathogensis of preeclampsia.
Selected Publications
Nold C, Anton L, Brown A, Elovitz MA: Inflammation promotes a cytokine response and disrupts the cervical epithelial barrier: a possible mechanism of premature cervical remodeling and preterm birth. American Journal of Obstetrics and Gynecology 2012 in press.Breen K, Burd I, Chai J, Friedman A, Elovitz MA: TLR4 dependent and independent mechanisms of fetal brain injury in the setting of preterm birth. Reproductive Sciences 2012 in press.
Bastek J, Elovitz M, McShea M, Foreman M Brown A, Adamczak J : The soluble receptor for advanced glycation end products (sRAGE) can prospectively identify patients at greatest risk for preterm birth The Journal of Maternal-Fetal & Neonatal Medicine 2012 accepted.
Anton L, Brown A, Parry S, Elovitz M : Lipopolysaccharide Induces Cytokine Production and Decreases Extravillous Trophoblast Invasion through a mitogen-activated protein Kinase Mediated Pathway: Possible Mechanisms of First Trimester Placental Dysfunction Human Reproduction [Epub ahead of print] November 2011
Elovitz M, Brown A, Breen K, Anton L, Maubert M, Burd I. : Intrauterine inflammation, insufficient to induce parturition, still evokes fetal and neonatal brain injury. International Journal of Neuroscience 29(6): 663-71, October 2011
Bastek JA, Adamczak JE, Elovitz MA, Hoffman S, Srinivas SK. : Trends in prematurity: What do changes at an urban institution suggest about the public health impact of 17-alpha hydroxyprogesterone caproate? Maternal and Child Health Journal.[Epub ahead of print] April 2011
Dalamaga M, Srinivas SK, Elovitz MA, Chamberland J, Mantzoros CS. : Serum adiponectin and leptin in relation to risk for preeclampsia: results from a large case-control study. Metabolism 60(11): 1539-44, November 2011.
Su HI, Schreiber CA, Fay C, Parry S, Elovitz MA, Zhang J, Shaunik A, Barnhart K. : Mucosal integrity and inflammatory markers in the female lower genital tract as potential screening tools for vaginal microbicides. Contraception. 84(5): 525-32, November 2011.
Calhoun KC, Barnhart KT, Elovitz MA, Srinivas SK: Evaluating the Association between Assisted Conception and the Severity of Preeclampsia. ISRN Obstet Gynecol.(928592), October 2011.
Burd I, Brown A, Gonzalez J, Chai J, Elovitz M. : A mouse model of term chorioamnionitis: unraveling causes of adverse neurological outcomes. Reproductive Sciences 18(9): 900-7, September 2011.

